EUROPEAN GENE ?
Update (see Understanding
There is a theory that
the mutation is recent
as a response to previous epidemics like the plague in Europe
but this would not explain
its clearly gradient north-south distribution
nor its limitation to Caucasians.
Gero Hüter and Susanne Ganepola write in The
CCR5-delta32 polymorphism as a model to study host adaptation against
infectious diseases and to develop new treatment strategies, Hütter
Biol Med (Maywood).
2011 Aug 1;236(8):938-43. Epub 2011 Jun 29
"The high allelic
frequency of CCR5-delta32 of 10–15% in Caucasians and the
misbalance in geographical distribution of this mutation is
suggestive to assume a beneficial role for the CCR5 depletion and
confers adaptation of the host toward (unknown) environmental threats."
We are guessing there is more to this
in terms of human genetics.
A European Gene
shows North-South Gradation
methods are an objective research path
for checking linguistic and archaeological theories
about the origin
and migration of ancient peoples and cultures.
LexiLine strongly supports the position that these modern genetic
methods will give us objective criteria to measure against
subjective academic opinions about ancient human history.
The August 7, 1998,
German daily, Die Welt,
contained an article by Susanne Horst
"Zehn Prozent der Europaeer sind vor Aids geschuetzt",
summarizing the genetic findings of the national cancer center
in Chicago as presented by molecular biologist Stephen J. O'Brien.
Human Gene Mutation
There is apparently
a human gene mutation,
which makes its holders nearly immune to AIDS,
since this gene has no receptor for AIDS-similar viruses.
inherited this gene from BOTH parents
is fairly immune to AIDS. Whoever has inherited this gene
from only ONE parent also has a good deal of immunity.
(The immunity is not perfect in either case, since rare strains
of AIDS can use the receptor CXCR 4).
A gene mutation is
generally a matter of chance
and its distribution among the population, according to O'Brien,
reaches only 1 percent at the most.
As the researchers
have discovered, however,
"Mutation CCR-5-delta-32" not only is immense in its distribution,
but it also has a distinct north to south distribution,
and, on the evidence of Australia, an "Indo-European" touch.
Here is the
percentage distribution by country
in percent of 100 percent
of the total population in that one country:
percent of all Swedes), Russia (13.6 of all Russians),
Estonia - [near Latvia] (13.3), Poland (13.3), Slovakia (13.3),
Australia (11.8), Great Britain (11.7), Ireland (11.3), Germany (10.8),
Czechoslovakia (10.2), Spain (9.8), Finland (9.1); France (8.9),
Austria (8.9), Denmark (8.3), Albania (8.2), Slovenia (7.7),
Turkey (6.3), Italy (5.5), Azerbaijan - southeast Caucasus (5.0),
Greece (4.4), Uzbekistan (3.4), Mexico (2.4).
have ZERO amounts of this gene:
Georgians in the
(thought to be related to the Basques),
Saudi Arabians (0), Cheyenne Indians (0), Pima Indians (0),
Pueblo Indians (0), Koreans (0) and mainland Chinese (0).
As the Chicago
researchers note, this gene mutation
is found only in white Europeans of Caucasian origin
- but not in persons of African, East-Asian or Indian origin.
Anthropologists think that the Caucasians were isolated
circa 200,000 years ago and that the mutation
must have occurred after this time.
O'Brien, by tracing the chemical evolution
of this gene back in terms of time, the mutation must be
around 127,500 years old, i.e. the era of emergence of "modern" man.
The north to south
of this gene indicates that the mutation
first took place in northern Europe
and then spread south slowly in the course of migrations,
but how would that fit the Out of Africa theory?
Or is this a Neanderthal remnant?
Why it is limited
is of course a cardinal question
is currently not known.
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